Epernicus: Jose M Trevejo, M.D., Ph.D.

Jose M Trevejo, M.D., Ph.D.

Positions:
Pricipal Biomedical Scientist

Biomedical Engineering

The Charles Stark Draper Laboratory (Cambridge, MA)

Clinical Attending

Medicine

Division of Infectious Disease

Beth Israel Deaconess Medical Center (Boston, MA)

Instructor in Medicine

Harvard Medical School (Boston, MA)

Co-Chair

Technology Working Group

Harvard Initiative for Global Health (Cambridge, MA)
Degrees:

M.D., Weill Cornell Medical College (New York, NY)

Ph.D., Molecular and Cellular Biology, Cornell University (Ithaca, NY)

B.Sc., Applied Mathematics, University of California, Los Angeles (Los Angeles, CA)
Clinical Training:

Fellowship, Infectious Disease, Beth Israel Deaconess Medical Center

Residency, Internal Medicine, Brigham and Women's Hospital
Past Advisors:

Lloyd Old (as Graduate Student - Ph.D.)

Erik Falck-Pedersen (as Graduate Student - Ph.D.)
Research:
Application of novel technologies to infectious disease and other global health problems.

Currently my work focuses on development and testing of novel technologies for implementation in rapid point-of-care diagnostics. My current projects are centered on the development of a novel sensor, the differential mobility spectrometer, for rapid diagnosis of tuberculosis and other pulmonary infectious diseases. I also continue to see patients in the Division of Infectious Disease at Beth Israel Deaconess Hospital.
Other Experience:
2007 Commercializing Science Course-Harvard Business School

Communities:

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TNF-alpha -dependent maturation of local dendritic cells is critical for activating the adaptive immune response to virus infection.
Trevejo JM, Marino MW, Philpott N, Josien R, Richards EC, Elkon KB, Falck-Pedersen E

Proceedings of the National Academy of Sciences of the United States of America. 2001 Oct 9;98(21):12162-7
Noggin antagonizes BMP signaling to create a niche for adult neurogenesis.
Lim DA, Tramontin AD, Trevejo JM, Herrera DG, García-Verdugo JM, Alvarez-Buylla A

Neuron. 2000 Dec;28(3):713-26
Inhibition of tumor necrosis factor alpha by an adenovirus-encoded soluble fusion protein extends transgene expression in the liver and lung.
Peng Y, Trevejo J, Zhou J, Marino MW, Crystal RG, Falck-Pedersen E, Elkon KB

Journal of virology. 1999 Jun;73(6):5098-109
Tumor necrosis factor alpha plays a central role in immune-mediated clearance of adenoviral vectors.
Elkon KB, Liu CC, Gall JG, Trevejo J, Marino MW, Abrahamsen KA, Song X, Zhou JL, Old LJ, Crystal RG, Falck-Pedersen E

Proceedings of the National Academy of Sciences of the United States of America. 1997 Sep 2;94(18):9814-9

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